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Dong H. Shin, MD, PhD
Detroit, Mich

Arch Ophthalmol. 1989;107(11):1566.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—We read with great interest the article by Johnson et al¹ in the May 1989 issue of the ARCHIVES. The authors list, as possible causes of the transient hypopyon in 2 of their 30 patients, whom they managed with pars plana vitrectomy and silicone oil injection for complex retinal detachment, an exaggerated inflammatory reaction possibly related to the intensive panretinal laser endophotocoagulation and an exaggerated inflammatory reaction possibly due to contaminants that might have been present in the silicone oil.

I would like to point out reports in the literature of three cases of transient hypopyon following argon laser photocoagulation of intraocular tissue.^2,3 All three cases of hypopyon followed intensive argon laser application to the iris for iridotomy, and two of the three cases were with documented rubeosis iridis associated with proliferative diabetic retinopathy. It seems, therefore, quite probable that the increased ocular inflammatory reaction . . . [Full Text PDF of this Article]

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