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Jeffrey T. Keller, OD
Birmingham, Ala

Arch Ophthalmol. 1973;90(6):511-512.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—Marmor has recently reported in the ARCHIVES (89: 419,1973) an interesting case of transient hyperopia in diabetes. In his comment on the refractive changes, Dr. Marmor correctly states that the refractive and accommodative changes in diabetes are poorly understood. A mechanism described by me in the American Journal of Optometry and Archives of American Academy of Optometry (50:108, 1973) explains the refractive changes that have been reported in diabetes. I will not elaborate the complete mechanism here; however, I think there is a simple explanation for the changes Dr. Marmor observed.

If we assume for the time being that the patient was essentially emmetropic upon admission to the hospital, ie, the hyperopia developed after treatment had begun, the hyperopia probably developed as a result of rehydration of the lens. This hydration would have occurred as a result of the osmotic imbalance created by the high concentrations . . . [Full Text PDF of this Article]

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